Title page for ETD etd-3998-181249


Type of Document Master's Thesis
Author Shenal, Brian Vincent
Author's Email Address bshenal@vt.edu
URN etd-3998-181249
Title The Dynamic Cerebral Laterality Effect: Group Differences in Hostility, Cardiovascular Regulation, and Sensory Recognition
Degree Master of Science
Department Psychology
Advisory Committee
Advisor Name Title
Harrison, David W. Committee Chair
Bell, Martha Ann Committee Member
Winett, Richard A. Committee Member
Keywords
  • Hostility
  • Lateralization
  • Stress
  • Asymmetry
  • Tachistoscope
  • Dichotic Listening
  • Cardiovascular Disease
Date of Defense 1998-04-08
Availability unrestricted
Abstract
THE DYNAMIC CEREBRAL LATERALITY EFFECT:

GROUP DIFFERENCES IN HOSTILITY, CARDIOVASCCULAR REGULATION,

AND SENSORY RECOGNITION

By Brian Vincent Shenal

David W. Harrison, Ph.D., Chairman

Psychology

(ABSTRACT)

This experiment tested two hypotheses linking the

right cerebral regulation of hostility and cardiovascular

arousal. First, replication of previous research

supporting heightened cardiovascular (systolic blood

pressure, diastolic blood pressure, and heart rate)

reactivity among high hostile participants was attempted.

Second, dynamic variations in functional cerebral asymmetry

in response to pain (cold pressor) and emotional linguistic

processing was measured.

Low- and high-hostile participants were identified

using the Cook Medley Hostility Scale (CMHS). All

participants completed either the negative affective verbal

learning test (Experiment 1) or the cold pressor paradigm

(Experiment 2). Cardiovascular measures (SBP, DBP, and HR)

were recorded and either dichotic listening procedures

(Experiment 1) or tachistoscopic lexical recognition

procedures (Experiment 2) were administered before and

after the stressor.

The primary finding of this research was greater left

cerebral activation (decreased cardiovascular reactivity)

following the dichotic phoneme listening and the

tachistoscopic lexical recognition tasks and greater right

cerebral activation following pain (cold pressor) and

emotionally linguistic (affective verbal learning)

stressors.

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